By Roni Shiloh, Rafael Stryjer, Abraham Weizman, David J. Nutt
The second one version of this unprecedented booklet offers a entire knowing of the mechanisms of motion all in favour of psychiatric pharmacotherapy. utilizing innovative vibrant double-page spreads, this unprecedented e-book provides cutting-edge info on all of the easy rules of psychiatric pharmacotherapy, abused ingredients, drug interactions, and therapy innovations.
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Mol Pharmacol 1984; 25: 113–122. 13. You dim MBH, Finberg JPM. New directions in monoamine oxidase A and B: selective inhibitors and substrates. Biochem Pharmacol 1990; 41: 155–162. 14. Greenshaw AJ. Neurotransmitter interactions in psychotropic drug action; beyond dopamine and serotonin. J Psychiatry Neurosci 2003; 28: 247–250. 15. Frey KA, Koeppe RA, Kilbourn MR. Imaging the vesicular monoamine transporter. Adv Neurol 2001; 86: 237–247. 16. Fleckenstein AE, Hanson GR. Impact of psychostimulants on vesicular monoamine transporter function.
J Clin Psychopharmacol 1995; 15: 399–408. 26. Guentert TW, Mayersohn M. Clinicalpharmacokinetic profile of moclobemide and its comparison with other MAOinhibitors. Rev Contemp Pharmacother 1994; 5: 19–34. 23 01 Chapter 1360 10/26/05 11:38 AM Page 24 Basic Principles of Psychiatric Pharmacotherapy 27. Harvey AT, Preskorn SH. Cytochrome P450 enzymes: interpretation of their interactions with selective serotonin re-uptake inhibitors: Part 1. J Clin Psychopharmacol 1996; 16: 273–278. 28. Henry JP, Botton D, Sagne C et al.
Escitalopram appears to be a well-tolerated and effective antidepressant. With a favorable side-effect profile, it appears to be well tolerated even in patients who are intolerant of other SSRIs. Escitalopram may possess safety advantages, in that it appears to have potential for fewer drug–drug interactions due to little effect on the CYP system, and is less highly protein-bound than other SSRIs. 7 Pindolol – 5-HT1A and b-adrenergic antagonist Supposed mode of accelerating and augmenting the antidepressant effect of SSRIs Pindolol Soma of presynaptic nerve 5-HT1A 5-HT-PMT SSRI Pindolol 5-HT1A Increased synaptic serotonin Postsynaptic nerve Legend due to (1) 5-HT1A blockade by pindolol and subsequent increased excitability of the serotonergic nerve and decreased inhibition of secretion of 5-HT from nerve terminal and (2) reuptake inhibition by the SSRI 5-HT-R Action potential 5-HT 5-HT1A Inhibition Serotonin Inhibitory somatodendritic serotonergic autoreceptor 5-HT-PMT Plasma membrane transporter for serotonin 5-HT-R Serotonergic receptors (any kind) 5-HT SSRI 38 Presynaptic nerve terminal Selective serotonin reuptake inhibitor 02 Chapter 1360 10/26/05 11:39 AM Page 39 Antidepressant drugs and mood stabilizers Notes about the scheme Some of the b-adrenergic receptor antagonists have a potential use in psychiatric pharmacotherapy.